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People with two copies of a mutation that protects against the HIV virus, known as CCR5-delta 32, are more likely to die before age 76 than individuals without the mutation, according to new research published today in Nature Medicine. The finding is further evidence that geneticist He Jiankui, who used the CRISPR-Cas9 gene editing tool to engineer this mutation in twin babies last year, acted irresponsibly and without a sufficient understanding of how the modification <a href=https://www.cups-stanley.ca>stanley cup</a> might affect the future health of his unwitting test subjects. The new finding also shows how difficult it will be to devise safe human gene therapies; sometimes, even seemingly good mutations窶敗uch as one that confers an immunity to HIV窶把an produce bad c <a href=https://www.stanleymug.us>stanley mug</a> onsequences. Around 11 percent of northern Europeans have the CCR5-delta 32 mutation, but its very rare in Asian populations. Previous studies have show <a href=https://www.cups-stanley.uk>stanley uk</a> n that the mutation, while conferring resistance to the HIV virus, appears to put people at greater risk from other infectious diseases. The mutation, for example, makes people four times more likely to die from the flu. As prior research and the new study shows, the absence of a fully functional CCR5 gene has a pronounced effect on the overall health of an individual. This gene, which codes for a protein found in immune cells, is exploited by the HIV virus as a gateway into cells. But CCR5 is involved in other important bodily processes as well. Here is a functional protein that we know has an effect in the
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